We all come in different shapes and sizes. Science has proven that every individual has a unique biochemistry that changes with genetics, food ingestion, environmental factors, and lifestyles choices. The constant change in our everyday lives has a profound affect on our ever-changing biochemistry due to individual choices and surroundings. As a result, a “one- size-fits-all” approach to health, fitness, and healthy living advice is redundant. BioPrint takes away the redundancy of the mundane “one-size-fits-all” approach and provides a biochemistry blueprint to fat loss and body composition modifications.

What is BioPrint?

BioPrint is world-renowned strength coach and functional medicine practitioner, Charles R. Poliquin’s cutting edge fat loss and health assessment that is designed to improve body composition rapidly, effectively, and safely. It is a scientific system that determines the serum concentrations of the most important hormones in your body without blood work. BioPrint is calculated by obtaining 14 site-specific skin fold measurements with body fat calipers. An individual’s regional fat storage at these specific sites is indicative of hormonal imbalances. It is a quick, painless, and non-invasive body fat assessment. When you know which hormones are imbalanced, you can correct it by using specific training, nutrition, supplementation, and lifestyle interventions.

The 14 site-specific skin fold measurements include the cheek, chin, pectoral, tricep, bicep, subscapular, midaxillary, suprailiac, umbilical, Australian, quadriceps, hamstrings, knee, and calf with each site correlating to a specific hormone. The site-specific measurements are then entered into the BioPrint software program and calculated by Coach Poliquin’s BioPrint algorithm that will identify the hormonal imbalances. By knowing a person’s BioPrint, we can optimize body composition and health by following site-specific protocols.

Hormones of Interest and Fat Storage

Testosterone Affects fat storage on the chest and triceps
Estrogen Affects fat storage on the hips and thighs
Cortisol Affects fat storage on the stomach
Growth Hormone Affects fat storage on the knees and calves
Thyroid Hormone Affects fat storage on the rib cage
Insulin Affects fat storage on the love handles and back

Gender differences in fat storage patterns are a fact that can be seen by simply observing the shape of an individual. Men tend to have an apple shape, i.e. relatively more visceral and intraperitoneal fat storage. Women tend to have a pear shape, i.e. relatively more gluteo-femoral fat storage.

Testosterone acts via the androgen receptor and stimulates lipolysis (fat burning). Testosterone combats the effects of cortisol, the stress hormone. Cortisol is the hormone with the strongest relation to fat distribution in both genders. Cortisol acts via the glucocorticoid receptor to stimulate lipoprotein lipase (LPL). LPL is an enzyme that makes fat cells ready for storage. The fat in between the organs in the abdomen is known as visceral adipocytes. Visceral adipocytes have more glucocorticoid receptors than other fat cells. Torso fat cells have fewer glucocorticoid receptors and subcutaneous fat cells on the rest of your body have even fewer. The fat cells on the hips and upper legs have the least glucocorticoid receptors.

What does this mean?

This means cortisol causes fat storage primarily on your abdomen, followed by the rest of your torso and only minimally on your lower body.

Testosterone is a cortisol antagonist. Meaning, testosterone and cortisol are in a constant battle for the activity of LPL. Cortisol stimulates LPL while testosterone inhibits it. This indicates that testosterone has a permissive effect on cortisol’s action. Cortisol will pile fat on your belly if there is not enough testosterone to stop it.

What testosterone is to men, estrogen is to women. In women, estrogen is the primary cortisol antagonist that reduces LPL activity. This is a dose-response effect, not just a permissive effect like that of testosterone in men. A woman with high levels of estrogen will store fat in the pear shape pattern. A woman with low levels of estrogen will have a more evenly spread fat distribution pattern with more fat on the abdomen and torso and less on the lower body.

Growth hormone has a shrinking effect on fat cells (lipolysis). Growth hormone is another cortisol antagonist that increases the anti-cortisol activity of testosterone in men and estrogen in women.

Insulin is often called the storage hormone. Insulin interacts strongly with LPL to store fatty acids from the blood in fat tissue. Since cortisol stimulates the production of LPL, insulin reinforces the effect of cortisol. High insulin levels can therefore lead to preferential fat storage around the midsection.

Get your BioPrint today and get started on your personalized plan to get rid of stubborn fat, contact BPC today.